Aging and glycation, what’s new? Alcoholic beverages and glycation: triple punishment.

Already responsible for over 200 diseases, alcohol is a booster of glycation and therefore of accelerated aging!

Obtained by fermenting plant sugars, then sometimes by distillation, alcoholic beverages are often very high in calories. Worst of all, some alcohol-based cocktails enriched with sugary drinks. Conversely, depending on the grape variety, some red wines contain very few calories.

For many consumers, alcohol is the main source of excessive sugar consumption, leading via glycation to accelerated aging and age-related diseases.

Numerous studies show that addictive alcohol consumption is a major factor in the etiology of cognitive decline and dementia.

A recent study shows that the mechanisms by which alcohol affects cognitive functions are linked to glycation via the activation of AGEs receptors, making alcohol an over-accelerator of glycation and therefore of aging (1).

Alcohol alone is responsible for over 200 diseases!

What’s more, many alcoholic beverages, with their high sugar content, fuel glycation and thus aging. Finally, alcohol accelerates glycation, making alcohol consumption a bomb toward accelerated aging and age-related diseases.

© AGE Breaker 04 2024

[Glycation is one of the major causes of aging. Resulting from the fixation of sugars on the proteins constituting the organism, glycation generates toxic compounds that cause cellular aging. Glycation is particularly involved in metabolic disorders, skin aging and cognitive decline.]
[AGE BREAKER, patented nutritional supplements, based on rosmarinic acid, recognized by aging specialists around the world for their properties to reverse the effects of glycation.]

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(1) : RODRÍGUEZ DE FONSECA, Fernando, MEDINA-PAZ, Francisco, SAPOZHNIKOV, Mira, et al. Plasma Concentrations of High Mobility Group Box 1 Proteins and Soluble Receptors for Advanced Glycation End-Products Are Relevant Biomarkers of Cognitive Impairment in Alcohol Use Disorder: A Pilot Study. Toxics, 2024, vol. 12, no 3, p. 190.