Eating slowly could considerably reduce the risk of metabolic syndrome and age-related diseases.
Time spent at the table varies greatly from country to country.
According to OECD statistics, France and several Mediterranean countries (Italy, Greece, Spain) are the world champions in terms of time spent eating, with more than 2 hours per day. Other European countries follow, along with Korea, China and Japan. The USA and Canada come last, with barely 1 hour a day spent eating (1).
Beware, these statistics may hide information concerning the destructuring of meals, a major cause of the rise in obesity: increased snacking and eating time spent in front of a screen, especially among the younger generations.
Eat fast, beware danger!
Japanese researchers have made an interesting discovery (2): by following 1,083 healthy people, aged 51 on average, over a 5-year period, they observed that eating fast causes significant fluctuations in glucose levels, leading to insulin resistance fuelled by glycation. This practice thus increases the risk of developing age-related diseases (diabetes, cardiovascular diseases) and accelerated aging.
Eating slowly, a weapon against aging:
The same studies show that slow eaters are 3 times lower risk to develop metabolic syndrome than normal eaters, and 6 times lower than fast eaters!
© AGE Breaker, updated 10 2024
[Glycation is one of the major causes of aging. Resulting from the fixation of sugars on the proteins constituting the organism, glycation generates toxic compounds that cause cellular aging. Glycation is particularly involved in metabolic disorders, skin aging and cognitive decline.] [AGE BREAKER, patented nutritional supplements, based on rosmarinic acid, recognized by aging specialists around the world for their properties to reverse the effects of glycation.]More on www.agebreaker.com
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(2) : YAMAJI, Takayuki, MIKAMI, Shinsuke, KOBATAKE, Hiroshi, et al. Slow down, you eat too fast: fast eating associate with obesity and future prevalence of metabolic syndrome. Circulation, 2017, vol. 136, no suppl_1, p. A20249-A20249.
Doi: 10.1161/circ.136.suppl_1.20249