Disturbed in its metabolism by an unsuitable lifestyle, glucose, essential fuel for the brain, would become one of the drivers of neuronal senescence.
Glucose is the main source of energy for the brain. Thus neurons, which neither produce nor store glucose, consume up to 25% of the energy from glucose. This implies a close and necessarily fragile coupling between energy demand and supply.
The overconsumption of sugars and in particular of fructose inevitably disrupts this mechanism.
Recent studies have shown that cognitive impairment in Alzheimer’s disease is associated with abnormal glucose metabolism (1).
This situation opens the way to metabolic reprogramming, combining diet and epigenetics… as a new strategy in the treatment of neuronal senescence and other diseases related to aging.
© AGE Breaker 10 2022
✅ [AGE BREAKER, patented nutritional supplements, based on rosmarinic acid, recognized by aging specialists around the world for their properties to reverse the effects of glycation.]
✅ [Glycation is one of the major causes of aging. Resulting from the fixation of sugars on the proteins constituting the organism, glycation generates toxic compounds that cause cellular aging. Glycation is particularly involved in metabolic disorders, skin aging and cognitive decline.]
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(1): Q. Wang et Al. Glucose Metabolism, Neural Cell Senescence and Alzheimer’s Disease. Int. J. Mol. Sci. 2022, 23(8),4351;
https://doi.org/10.3390/ijms23084351
